Bio
We haven't found any bio for you yet.
Researcher Links
Loading links...
Publications by Type
Loading publications…
The last 5 uploaded publications
State of play and future direction with NOACs: An expert consensus
Alexander T. Cohen, Gregory Y.H. Lip, R De Caterina, Hein Heidbüchel, José Luis Zamorano, Giancarlo Agnelli, Freek W.A. Verheugt, A. John Camm (2018). State of play and future direction with NOACs: An expert consensus. , 106, DOI: https://doi.org/10.1016/j.vph.2018.04.001.
Article290 days agoP1062 Dilated cardiomyopathy caused by lamin A/C mutation: need for a prospective study to prevent sudden death in lamin A/C mutation carriers
Tom Rossenbacker, YM Pinto, Johan Van Cleemput, Koenraad Devriendt, J. Peter van Tintelen, Peter Carmeliet, Hein Heidbüchel, Jop H. van Berlo (2003). P1062 Dilated cardiomyopathy caused by lamin A/C mutation: need for a prospective study to prevent sudden death in lamin A/C mutation carriers. , 24(5), DOI: https://doi.org/10.1016/s0195-668x(03)94354-5.
Article171 days agoUnconventional intronic splice site mutation in SCN5A associates with cardiac sodium channelopathy
Tom Rossenbacker, Els Schollen, Cuno Kuipéri, Thomy de Ravel, Koenraad Devriendt, Gert Matthijs, Désiré Collen, Hein Heidbüchel, Peter Carmeliet (2005). Unconventional intronic splice site mutation in SCN5A associates with cardiac sodium channelopathy. , 42(5), DOI: https://doi.org/10.1136/jmg.2004.029058.
Article171 days agoNovel pore mutation in SCN5A manifests as a spectrum of phenotypes ranging from atrial flutter, conduction disease, and Brugada syndrome to sudden cardiac death
Tom Rossenbacker, Sheila J. Carroll, Huajun Liu, Cuno Kuipéri, Thomy de Ravel, Koenraad Devriendt, Peter Carmeliet, Robert S. Kass, Hein Heidbüchel (2004). Novel pore mutation in SCN5A manifests as a spectrum of phenotypes ranging from atrial flutter, conduction disease, and Brugada syndrome to sudden cardiac death. , 1(5), DOI: https://doi.org/10.1016/j.hrthm.2004.07.001.
Article171 days agoUnconventional splice site mutation in SCN5A as cause (and modifier?) of overlapping phenotypes of Brugada syndrome and isolated cardiac conduction defect
Tom Rossenbacker, Els Schollen, Cuno Kuipéri, Thomy de Ravel, Gert Matthijs, Hein Heidbüchel, Peter Carmeliet (2004). Unconventional splice site mutation in SCN5A as cause (and modifier?) of overlapping phenotypes of Brugada syndrome and isolated cardiac conduction defect. , 110(17)
Article171 days ago