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Get Free AccessCholinergic stimulation provokes arrhythmias in this model of LQT3 by triggering bradycardia. beta-Adrenoceptor density is reduced, and beta-adrenoceptor blockade does not prevent arrhythmias. Sodium channel blockade and beta-adrenoceptor stimulation suppress arrhythmias by shortening repolarization and minimizing difference in late sodium current.
Larissa Fabritz, Dierk Damke, Markus Emmerich, Susann G. Kaufmann, Kathrin Theis, Andreas Blana, Lisa Fortmüller, Sandra Laakmann, Sven Hermann, Elena Aleynichenko, Johannes Steinfurt, Daniela Volkery, Burkhard Riemann, Uwe Kirchhefer, Michael R. Franz, Günter Breithardt, Edward Carmeliet, Michael Schäfers, Sebastian K.G. Maier, Peter Carmeliet, Paulus Kirchhof (2010). Autonomic modulation and antiarrhythmic therapy in a model of long QT syndrome type 3. , 87(1), DOI: https://doi.org/10.1093/cvr/cvq029.
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Type
Article
Year
2010
Authors
21
Datasets
0
Total Files
0
Language
en
DOI
https://doi.org/10.1093/cvr/cvq029
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